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Literaturverzeichnis

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116

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Congenital hypokalaemia with hypercalciuria in preterm infants: A hyperprostaglandinuric

tubular syndrome different from Bartter syndrome

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Seyberth HW, Königer SJ, Rascher W, Kühl PG, Schweer H

Role of prostaglandins in hyperprostaglandin E syndrome and in selected renal tubular

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Pediatr. Nephrol. 1: 491-497, 1987

Seyberth HW, Leonhardt A, Soergel M

Das Hyperprostaglandin-E

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Seyberth HW, Soergel M, Köckerling A

Hypokalaemic tubular disorders: the hyperprostaglandin E syndrome and Bartter syndrome

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nephrology, 2. ed.

Oxford University Press, Oxford, 1085-1094, 1998

Shoemaker L, Welch TL, Bergstrom W, Abrams A, Yergey AL, Vieira N

Calcium kinetics in the hyperprostaglandine E syndrome

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Shuck ME, Bock JH, Benjamin CW, Tsai TD, Lee KS, Slightom JL, Bienowski MJ

Cloning and characterization of multiple forms of the human kidney ROM-K potassium channel

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Simon DB, Nelson-Williams C, Bia MJ, Ellison D, Karet FE, Molina AM, Vaara I, Iwata F,

Cushner HM, Koolen AM, Gainza FJ, Gitelman HJ, Lifton RP

Gitelman`s variant of Bartter`s syndrome, inherited hypokalaemic alkalosis, is caused by

mutations in the thiazide-sensitive Na-Cl cotransporter

Nature Gen. 12: 24-30, 1996a

Simon DB, Karet FE, Hamdan JH, DiPietro A, Sanjad SA, Lifton RP

Bartter`s syndrome, hypokalaemic alkalosis with hypercalciuria, is caused by mutations in the

Na-K-Cl cotransporter NKCC2

Nature Gen. 13: 183-188, 1996b

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Wolf K45E Bedienungsanleitung Seite 123